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Tuesday, July 28, 2020 | History

4 edition of SAM model of senescence found in the catalog.

SAM model of senescence

proceedings of the first International Conference on Senescence, the SAM model, Kyoto, 17-18 March 1994

by International Conference on Senescence (1st 1994 Kyoto, Japan).

  • 152 Want to read
  • 27 Currently reading

Published by Excerpta Medica in Amsterdam, New York .
Written in English

    Subjects:
  • Aging -- Animal models.,
  • Mice -- Diseases.,
  • Mice as laboratory animals.,
  • Older people -- Diseases -- Animal models.

  • Edition Notes

    Includes index.

    Statementeditor, Toshio Takeda.
    SeriesInternational congress series ;, no. 1062
    ContributionsTakeda, Toshio, 1931-
    Classifications
    LC ClassificationsQP86 .I579 1994
    The Physical Object
    Paginationxv, 458 p. :
    Number of Pages458
    ID Numbers
    Open LibraryOL1095853M
    ISBN 10044481695X
    LC Control Number94020411

      The model: the senescence-accelerated mouse (SAM) • Model of spontaneous senescence that displays many common geriatric disorders in human population • Two series: SAMR and SAMP • Breeders retrospectively chosen based on degree of senescence at eight months – Life span – Clinical signs of aging • Earlier onset and irreversible. ISBN: OCLC Number: Description: 1 online resource (xii, pages): illustrations (some color) Contents: Detecting cellular senescence in reprogramming / Coralie Cazin, Mathieu von Joest, and Han Li --DNA damage in situ ligation followed by proximity ligation assay (DI-PLA) / Alessandro Galbiati and Fabrizio d'Adda di Fagagna --Reactive oxygen species.

      Takeda T., Hosokawa M. and Higuchi K. () Senescence-accelerated mouse (SAM): a novel murine model of senescence. Exp. Gerontol. 32, – /S(96) [Google Scholar] 6. Morley J.E., Kumar Author: Zhengcai Du, Fangcao Fanshi, Yu-Heng Lai, Jung-Ren Chen, Erwei Hao, Jiagang Deng, Chung-Der Hsiao. The Biology of Senescence 3rd Edition by Alexander Comfort (Author) ISBN ISBN Why is ISBN important? ISBN. This bar-code number lets you verify that you're getting exactly the right version or edition of a book. The digit and digit formats both work. Format: Hardcover.

    SAM addresses project roadblocks (product quality, meeting timelines and budgets, and managing SMEs). Most importantly, SAM is an agile elearning development process built specifically to create performance-driven learning. The Agile Advantage. With SAM, your project starts at performance so that you end with g: senescence. Online Books; Nomenclature Home Page; Utani A., Umezawa M., and Takeda T. () Age-related changes in bone mass in the senescence accelerated mouse (SAM): SAM-R/3 and SAM-P/6 as new murine models for senile osteoporosis. and Higuchi K. () Senescence-accelerated mouse (SAM): A novel murine model of accelerated senescence. J. Amer.


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SAM model of senescence by International Conference on Senescence (1st 1994 Kyoto, Japan). Download PDF EPUB FB2

The Sam Model of Senescence: Proceedings of the First International Conference on Senescence: The Sam Model, Kyoto, March (International Congress Series) 1st Edition by Japan) International Conference on Senescence (Kyoto (Author), Toshio Takeda (Editor). The Senescence-Accelerated Mouse (SAM): An Animal Model of Senescence: Proceedings of the 2nd International Conference on Senescene: The SAM Model, ) (International Congress (Volume )): Medicine & Health Science Books @ The Senescence-Accelerated Mouse (SAM) has been under development by our research team at Kyoto University since through the selective inbreeding of the AKR/J strain of mice donated try the Jackson Laboratory inbased on a graded score for senescence Cited by: The senescence-accelerated mouse (SAM) model was established from the AKR/J strain as a spontaneous murine model of accelerated senescence.

Several studies have indicated that long-term administration of dietary bioactive components prevents learning and memory impairment in. Part 1 Animal models for research on ageing: comparative perspectives on phenotypic plasticity in the SAM mouse strains and other animals, C.E. Finch; senescence accelerated mouse (SAM); a novel murine model of ageing, T.

Takeda et al. Part 2 Topics in SAM research: murine senile amyloidosis and accelerated senescence, H. Naiki; molecular genetic studies on SAM, K. Higuchi. A novel murine model of aging, Senescence-Accelerated Mouse (SAM) Author links open overlay panel Toshio Takeda a Masanori Hosokawa a Keiichi Higuchi a Masamichi Hosono b Ichiro Akiguchi c Cited by: The P series was named Senescence-Accelerated Mouse (SAM), and our first article on the SAM model appeared in Mechanisms of Ageing and Development in (Takeda et al., ).

At that time, the mean life span in the P series was months, 27% shorter than that of the R series ( months).Cited by: The senescence-accelerated mouse (SAM), consisting of 14 senescence-prone inbred strains (SAMP) and 4 senescence-resistant inbred strains (SAMR) has been under development since through the selective inbreeding of AKR/J strain mice donated by the Jackson laboratory inbased on the data of the grading score of senescence, life span, and pathologic by: The Senescence-Accelerated Mouse (SAM) represents a group of inbred mouse strains developed by Toshio Takeda and his colleagues at Kyoto University as a model for the study of human aging and age-related by: Introduction An animal model for accelerated senescence, which we named Senescence- Accelerated Mouse (SAM), was developed in the Department of Senescence Biology (formerly the Department of Pathology), Chest Disease Research Institute, Kyoto University, ~ginning in Cited by: Author summary The senescence associated secretory phenotype is developed by cells undergoing permanent cell cycle arrest.

This phenotype is characterized by the secretion of a variety of factors that facilitate tissue breakdown and inflammation and is therefore theorized to, in part, be causal for aging and age-related diseases. In recent years the SASP has been implicated in a variety of Cited by:   The Successive Approximation Model or SAM is the latest and greatest proposal in Instructional Systems Design (ISD) getting significant fanfare by the American Society of Training and Development (ASTD).Missing: senescence.

Analysis of the oxidative stress state in the brain and peripheral organs of senescence-accelerated mouse (SAM) model (S. Matsugo, F. Yasui, K. Sasaki). Mitochondrial alterations and a higher oxidative status in cultured fibroblast-like cells from senescence-accelerated mice (Y. Chiba et al.).

SAM is a very promising model for studying the relationships of body aging and cell senescence in by: 9. The SAM strains, a group of related inbred strains consisting of senescence-prone inbred strains (SAMP) and senescence-resistant inbred strains (SAMR), have been successfully developed by selective inbreeding of the AKR/J strain of mice donated by the Jackson laboratory in The characteristic feature of aging common to the SAMP and SAMR is accelerated senescence and Cited by: Cite this chapter as: Yagi H., Akiguchi I., Takeda T.

() Senescence-Accelerated Mouse SAM-P/8 Shows Spontaneous Age-Related Impairment of Ability of Acquisition of Learning and Memory: An Animal Model of Disturbances in Recent Memory with : Hideo Yagi, Ichiro Akiguchi, Toshio Takeda. Chapters focus on the high heterogeneity of the senescence phenotypes, and techniques to induce and identify specific senescence programs.

Additional chapters describe cellular and mouse models in which is possible to study the complex cell and non-cell autonomous functions of senescent cells. The senescence-accelerated mou se (SAM) is a group of inbred mouse strains that are u sed as animal models of senescence acceleration and age-associated disorders.

It is known that the longevity of senescence accelerated mouse (SAM) is significantly reduced. We intended to check the relationship of the SAM shortened longevity with some characteristics of thei Cited by: 9. Introduction. The development of a senescence-accelerated mouse (SAM 1) strain has been under way at Kyoto University since At present, 12 lines of inbred strains have been maintained as original colonies of the SAM model: 9 senescence-prone (SAMP 1) and 3 senescence-resistant (SAMR 1).In addition, several lines of SAM have been developed at Takeda Chemical Ind., Cited by:.

The Senescence-Accelerated Mouse (SAM) strain was established in the Department of Senescence Biology, Chest Disease Research Institute, Kyoto University, as a novel murine model of senescence acceleration and age-associated by: Generally, rats and mice are mostly used as animal models in skeletal muscle ageing research.

Senescence-Accelerated Mouse (SAM), which consists of 18 lines: 11 senescence-prone inbred strains (SAMP) and 7 senescence-resistant inbred strains (SAMR), are regarded as a good choice for sarcopenia study [28, 29].Cited by: DeepDyve is the largest online rental service for scholarly research with thousands of academic publications available at your fingertips.